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Bath 2:20, 5f Maiden (6) |
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| Horse | SP | [EST] | LB | Run | Wnr | Shp | Trainer | Dw | Hf | LBL | Run Notes | ||||
| 1 | King Crimson | 3 | 50.0 | - | 6 | N | M. R. Channon | 7 | 0.0 | Biggest, Dev Ok from Early Season | |||||
| 2 | Free Entry | 0.33 | 44.8 | 0.8 | 4 | M | J. J. S. Tate | 8 | 0.0 | Supp 8/15, Smaller Mdm, Lack Scope, RcCent & JkActv Ov, , WTRB = Vet said Hrs suffering exertional rhabdomyolysis.post-race. | |||||
| 3 | Dunnscotia (t) | 6 | 37.8 | 4.8 | 3 | P. R. Webber | 1 | 0.0 | Supp 7/1, Fizzy AP, Stroppy & TblLdg, Held & Hooded Stalls, , Smaller & Nrrwr than KC | ||||||
| 4 | Cape Point | 25 | 34.8 | 5.8 | 3 | M. Blanshard | 6 | 0.0 | |||||||
| 5 | King's Bond | 20 | 16.8 | 11.8 | 3 | R. Harris | 2 | 0.0 | OkBld but Small & Short | ||||||
| 6 | My Mo | 25 | 15.3 | 12.3 | 2 | D. R. Dennis | 5 | 0.0 | UnsttlStalls, | ||||||
| 7 | Cerise Firth | 33 | 8.8 | 12.8 | 4 | S. Hollinshead | 3 | 0.0 | |||||||
| 8 | Blue Eyed Boy (b) | 33 | -124.3 | 58.8 | 4 | R. Brotherton | 4 | 0.0 | , WTRB = Jk said Saddle Slipped, Vet said Hrs Lame on OF post-race | ||||||
| WD | John Joiner | P. J. Makin | 9 | WD = Unruly on JU in Pre-Parade & Jk unable to get Mounted. | |||||||||||
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F from GFF on Decs. Stiff- WSW Wind = 1/2 Headwind from Left then 3/4 Headwind last 2F. 10p/£ Rule 4 Deduction. Ragged and somewhat chaotic race overall with a lot of poor behaviour beforehand and a stiff enough pace to put the garbage under pressure early and the leading 4 never making a proper 'Group', Problems continued Post-Race with 2 x Vet's Reports. Free Entry never travelling well racing centre and looking a bit unbalanced 1f out but finding some laboured rolling on for a hard ride in the last 1f. Vet reported she had 'exertional rhabdomyolysis.' post race. Wikipedia struggles to come up with a simple summary of what this is. For example uder 'Physiology' it records this = "Rhabdomyolsis causes the myosin and actin to degenerate into smaller proteins that travel into the circulatory system. The body reacts by increasing intracellular swelling to the injured tissue to send repair cells to the area. This allows creatine kinase and myoglobin to be flushed from the tissue where it travels in the blood until reaching the kidneys.[9] In addition to the proteins released large quantities of ions such as intracellular potassium, sodium, and chloride find their way into the circulatory system. Intracellular potassium ion has deleterious effects on the heart's ability to generate action potentials leading to cardiac arrhythmias. Consequently, this can affect peripheral and central perfusion that can affect all major organ systems in the body.". An overall summary would be the 'Breakdown of Muscle fibres from extreme Physical exertion'. The body not being able to use available energy sources starts breaking down the available muscle proteins. This causes a range of physical & mechanical knock-on issues. Under 'Causes' it notes = .ER is more likely to occur when strenuous exercise is performed under high temperatures and humidity. Poor hydration levels before, during, and after strenuous bouts of exercise have also been reported to lead to ER. This condition and its signs and symptoms are not well known amongst the sport and fitness community and because of this it is believed that the incidence is greater but highly underreported. Risks that lead to ER include exercise in hot and humid conditions, improper hydration, inadequate recovery between bouts of exercise, intense physical training, and inadequate fitness levels for beginning high intensity workouts. Dehydration is one of the biggest factors that can give almost immediate feedback from the body by producing very dark colored urine.". It was around 15DegC at BATH on the day. |